Ozone: a possible cause of hemolytic anemia in glucose-6-phosphate dehydrogenase deficient individuals
Calabrese EJ, Kojola WH, Carnow BW. Journal of Toxicology and Environmental Health, 1977.
- Study type
- Other
- Published
- 1977, Journal of Toxicology and Environmental Health
- Identifiers
- PMID 846014, DOI 10.1080/15287397709529470
What the authors reported
A theoretical model predicting that people with glucose-6-phosphate dehydrogenase (G6PD) deficiency may experience acute hemolysis on exposure to ozone at levels reached in polluted urban air.
What limits this result
A theoretical, computational model of inhaled ambient ozone, not an experiment on blood or on any ozone therapy procedure; does not test the brief controlled blood contact EBO2 or MAH use.
Harms reported after ozone therapy, and studies of how ozone exposure affects health. One study rarely settles a medical question; our guides weigh each paper against the rest of the evidence.
How the study was done
- Design
- Theoretical model
- Read from
- Read from the abstract only; where this page says nothing about an item, the abstract does not report it. Read on September 28, 2026.
Source: Abstract on PubMed
What it measured
- Model prediction for people with glucose-6-phosphate dehydrogenase deficiency exposed to ozone
- Predicts that individuals with a glucose-6-phosphate dehydrogenase deficiency may experience acute hemolysis on exposure to ozone at levels reached in certain urban centers
Source: Abstract on PubMed
Where we cite this study
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